Researchers at University of California San Diego School of Medicine have discovered a previously unrecognized mechanism by which Staphylococcus aureus—one of the most common causes of skin and soft tissue infections worldwide—delays wound healing. The new study reveals that quorum sensing—a process in which bacteria communicate and coordinate behavior with one another—is a key driver of delayed healing in wounds infected by S. aureus. The findings suggest that by using drugs to interfere with quorum sensing, it could be possible to enhance wound healing without relying on antibiotics, reducing the risk of resistance and improving healing outcomes for patients.
Bacterial communication delays wound healing by disrupting skin cell repair
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